Hasn’t had an attack in several years!

“How on earth do you find all these remedies?” I asked them. “Actually,” they replied, “some of the best ones find us! For example…

“A woman phoned in to our radio show with a wild-sounding asthma remedy. She’d been suffering for years, but she said her life has been totally changed by…

WILD CHERRY-BARK TEA! “She swore she hasn’t had an asthma attack since she started drinking it 5 years ago. Still, we wondered…

“WILL IT WORK FOR ANYONE ELSE?”

“Does it ever! Ever since that radio show, folks with breathing problems have been telling us THANK YOU! They say it’s great for asthma, bronchitis and coughs.”

NOTE: This unusual tea can be hard to find.

Sourced & published by Henry Sapiecha

Optical Technology Shows Potential

for Prescreening Patients at High Risk

Science (Oct. 9, 2010) — Researchers from Northwestern University and NorthShore University HealthSystem (NorthShore) have developed a method to detect early signs of lung cancer by examining cheek cells in humans using pioneering biophotonics technology.

Early detection is critical for improving cancer survival rates. Yet, one of the deadliest cancers in the United States, lung cancer, is notoriously difficult to detect in its early stages. Now, researchers have developed a method to detect lung cancer by merely shining diffuse light on cells swabbed from patients’ cheeks.

“By examining the lining of the cheek with this optical technology, we have the potential to prescreen patients at high risk for lung cancer, such as those who smoke, and identify the individuals who would likely benefit from more invasive and expensive tests versus those who don’t need additional tests,” said Hemant K. Roy, M.D., director of gastroenterology research at NorthShore.

The optical technique is called partial wave spectroscopic (PWS) microscopy and was developed by Vadim Backman, professor of biomedical engineering at Northwestern’s McCormick School of Engineering and Applied Science. Backman and Roy earlier used PWS to assess the risk of colon and pancreatic cancers, also with promising results.

The lung cancer findings are published online Oct. 5 by the journal Cancer Research. The paper will appear in print in the Oct. 15 issue.

Lung cancer is the leading cause of cancer deaths in the United States. Survival rates are high with surgical resection (removal of the tumor) but only if detected at an early stage. Currently there are no recommended tests for large population screening to detect lung cancer early. The disease is already advanced by the time most lung cancer patients develop symptoms. The five-year survival rate for lung cancer patients is only 15 percent.

PWS can detect cell features as small as 20 nanometers, uncovering differences in cells that appear normal using standard microscopy techniques. The PWS-based test makes use of the “field effect,” a biological phenomenon in which cells located some distance from the malignant or pre-malignant tumor undergo molecular and other changes.

“Despite the fact that these cells appear to be normal using standard microscopy, which images micron-scale cell architecture, there are actually profound changes in the nanoscale architecture of the cell,” Backman said. “PWS measures the disorder strength of the nanoscale organization of the cell, which we have determined to be one of the earliest signs of carcinogenesis and a strong marker for the presence of cancer in the organ.”

“PWS is a paradigm shift, in that we don’t need to examine the tumor itself to determine the presence of cancer,” added Hariharan Subramanian, a research associate in Backman’s lab who played a central role in the development of the technology.

After testing the technology in a small-scale trial, Roy and Backman focused the study on smokers, since smoking is the major risk factor related to 90 percent of lung cancer patients. “The basic idea is that smoking not only affects the lungs but the entire airway tract,” Roy said.

The study was comprised of 135 participants including 63 smokers with lung cancer and control groups of 37 smokers with chronic obstructive pulmonary disease (COPD), 13 smokers without COPD and 22 non-smokers. The research was not confounded by the participants’ demographic factors such as amount of smoking, age or gender. Importantly, the test was equally sensitive to cancers of all stages, including early curable cancers.

The researchers swabbed the inside of patients’ mouths, and then the cheek cells were applied to a slide, fixed in ethanol and optically scanned using PWS to measure the disorder strength of cell nanoarchitecture. Results were markedly elevated (greater than 50 percent) in patients with lung cancer compared to cancer-free smokers.

A further assessment of the performance characteristics of the “disorder strength” (as a biomarker) showed greater than 80 percent accuracy in discriminating cancer patients from individuals in the three control groups.

“The results are similar to other successful cancer screening techniques, such as the pap smear,” Backman said. “Our goal is to develop a technique that can improve the detection of other cancers in order to provide early treatments, much as the pap smear has drastically improved survival rates for cervical cancer.”

Additional large-scale validation trials are necessary for PWS. If it continues to prove effective in clinical trials at detecting cancer early, Backman and Roy believe PWS has the potential to be used as a prescreening method, identifying patients at highest risk who are likely to benefit from more comprehensive testing such as bronchoscopy or low-dose CT scans.

The paper is titled “Optical Detection of Buccal Epithelial Nanoarchitectural Alterations in Patients Harboring Lung Cancer: Implications for Screening.” In addition to Roy, Backman and Subramanian, other authors of the paper are Dhwanil Damania, Thomas A. Hensing, William N. Rom, Harvey I. Pass, Daniel Ray, Jeremy D. Rogers, Andrej Bogojevic, Maitri Shah, Tomasz Kuzniar and Prabhakar Pradhan.

Editor’s Note: This article is not intended to provide medical advice, diagnosis or treatment.

Sourced & published by Henry Sapiecha

HONG KONG, Oct. 7, 2010 (Reuters) — Some Chinese cigarettes contain amounts of lead, arsenic and cadmium that are three times higher than levels found in Canadian cigarettes, a study has found.

While consuming such heavy metals is widely known to be harmful to health, there is little research done so far about their impact when inhaled into the body.

The researchers, who published their findings in the journal Tobacco Control on Thursday, said more investigation was needed.

“While the per-stick levels of metals are what we measured, the real issue is repeated exposure. Smokers don’t smoke just one cigarette, but 20 or so a day every day for years because cigarettes are addictive,” wrote lead author Richard O’Connor of the Roswell Park Cancer Institute in Buffalo, New York.

“These metals get into smokers along with a cocktail of other toxicants. The effect of cumulative exposure to multiple toxicants, including metals, is the public health question that needs to be sorted out.”

The researchers used Canadian cigarettes for comparison in their study because Canadian manufacturers and importers are required to test for metals content in tobacco, and Health Canada, the country’s public health agency, recently released data concerning this.

China has more than 320 million smokers and a million Chinese in the country die each year from tobacco-related illnesses. Smoking has been causally linked to hypertension, stroke, diabetes, cancer, heart and respiratory diseases, among others.

China has the world’s largest smoking population and is also the biggest producer of tobacco, manufacturing 2.16 trillion cigarettes in 2007, according to the Tobacco Atlas.

O’Connor and colleagues analyzed 78 varieties of popular Chinese cigarette brands and found significantly elevated levels of heavy metals, with some containing three times the levels of lead, cadmium and arsenic compared with Canadian cigarettes.

“The higher yields of cadmium and lead in cigarettes manufactured in China are worrisome given current smoking prevalence in China and China National Tobacco Company’s export ambitions,” the researchers wrote in their paper.

A member of the team, Geoffrey Fong from the University of Waterloo in Canada, said the heavy metals content was due to contaminated soil.

“Tobacco like other crops absorbs minerals and other things from the soil, so if the soil has cadium, lead or arsenic, they will be absorbed into the tobacco,” Fong said.

Sourced & published by Henry Sapiecha

Using ‘Skeletons’ of Old Ones

Science (June 28, 2010) — For someone with a severe, incurable lung disorder such as cystic fibrosis or chronic obstructive pulmonary disease, a lung transplant may be the only chance for survival. Unfortunately, it’s often not a very good chance. Matching donor lungs are rare, and many would-be recipients die waiting for the transplants that could save their lives.

Such deaths could be prevented if it were possible to use stem cells to grow new lungs or lung tissue. Specialists in the emerging field of tissue engineering have been hard at work on this for years. But they’ve been frustrated by the problem of coaxing undifferentiated stem cells to develop into the specific cell types that populate different locations in the lung.

Now, researchers from the University of Texas Medical Branch at Galveston have demonstrated a potentially revolutionary solution to this problem. As they describe in an article published electronically ahead of print by the journal Tissue Engineering Part A, they seeded mouse embryonic stem cells into “acellular” rat lungs — organs whose original cells had been destroyed by repeated cycles of freezing and thawing and exposure to detergent.

The result: empty lung-shaped scaffolds of structural proteins on which the mouse stem cells thrived and differentiated into new cells appropriate to their specific locations.

“In terms of different cell types, the lung is probably the most complex of all organs — the cells near the entrance are very different from those deep in the lung,” said Dr. Joaquin Cortiella, one of the article’s lead authors. “Our natural matrix generated the same pattern, with tracheal cells only in the trachea, alveoli-like cells in the alveoli, pneumocytes only in the distal lung, and definite transition zones between the bronchi and the alveoli.”

Such “site-specific” cell development has never been seen before in a natural matrix, said professor Joan Nichols, another of the paper’s lead authors. The complexity gives the researchers hope that the concept could be scaled up to produce replacement tissues for humans — or used to create models to test therapies and diagnostic techniques for a variety of lung diseases.

“If we can make a good lung for people, we can also make a good model for injury,” Nichols said. “We can create a fibrotic lung, or an emphysematous lung, and evaluate what’s happening with those, what the cells are doing, how well stem cell or other therapy works. We can see what happens in pneumonia, or what happens when you’ve got a hemorrhagic fever, or tuberculosis, or hantavirus — all the agents that target the lung and cause damage in the lung.”

The researchers have already begun work on large-scale experiments, “decellularizing” pig lungs with an eye toward using them to produce larger samples of lung tissue that could lead to applications in humans. They’re also taking on the challenge of vascularization — stimulating the growth of blood vessels that will enable the engineered tissues to survive outside the special bioreactors that the researchers now use to keep them alive by bathing them in a life-sustaining cocktail of nutrients and oxygen.

“People ask us why we’re doing the lung, because it’s so hard,” Cortiella said. “But the potential is so great, and the technology is here. It’s going to take time, but I think we’re going to create a system that works.”

Other authors of the Tissue Engineering Part A paper are UTMB research associate Jean Niles, associate professor Gracie Vargas, medical student Sean Winston, graduate student Shannon Walls, summer research fellows Andrea Brettler and Jennifer Wang, Andrea Cantu of Stanford University and Dr. Anthony Pham of Brown Medical School

Sourced & published by Henry Sapiecha

Science (Oct. 7, 2008) — Moderate consumption of red wine may decrease the risk of lung cancer in men, according to a report in the October issue of Cancer Epidemiology, Biomarkers & Prevention¸ a journal of the American Association for Cancer Research.

“An antioxidant component in red wine may be protective of lung cancer, particularly among smokers,” said Chun Chao, Ph.D., a research scientist at Kaiser Permanente Department of Research and Evaluation in Pasadena, California.

Chao analyzed data collected through the California Men’s Health Study, which linked clinical data from California’s health system with self-reported data from 84,170 men aged 45 to 69 years. Researchers obtained demographics and lifestyle data from surveys computed between 2000 and 2003, and identified 210 cases of lung cancer.

Researchers measured the effect of beer, red wine, white wine and liquor consumption on the risk of lung cancer. Adjustments were made for age, race/ethnicity, education, income, body mass index, history of chronic obstructive pulmonary disease or emphysema, and smoking history.

Among the study participants, there was on average a two percent lower lung cancer risk associated with each glass of red wine consumed per month. The most substantial risk reduction was among smokers who drank one to two glasses of red wine per day. The researchers reported a 60 percent reduced lung cancer risk in these men. Researchers warned men to stop smoking as the best way to reduce lung cancer risk; noting that even men who drank one to two glasses of red wine per day still face higher lung cancer risk than do non-smokers.

No clear associations with lung cancer were noted for consumption of white wine, beer, or liquor. “Red wine is known to contain high levels of antioxidants. There is a compound called resveratrol that is very rich in red wine because it is derived from the grape skin. This compound has shown significant health benefits in preclinical studies,” Chao said.

Chao said their findings should not be construed to recommend heavy alcohol consumption.

Sourced & published by Henry Sapiecha

BETHESDA, Md. (UPI) — The U.S. National Institute of Allergy and Infectious Diseases says incidents of non-tuberculosis mycobacteria lung disease are increasing across the nation.

Researchers said non-tuberculous mycobacteria are environmental organisms found in both water and soil that can cause severe pulmonary disease in humans — and a large study indicates the disease is increasing.

A research team led by epidemiologists from the National Institute of Allergy and Infectious Diseases, part of the National Institutes of Health, analyzed hospital discharge records of patients in 11 states whose combined total population represents 42 percent of the country. They said they reviewed database records spanning 1998 to 2005 and identified more than 16,475 hospitalizations associated with non-tuberculosis mycobacteria in people without AIDS.

Before the widespread availability of combination antiretroviral therapy, pulmonary disease was a common opportunistic infection among people with AIDS. The study was limited to patients not suffering from AIDS.

Researchers said of the 11 states studied, Florida, New York and California had 62 percent of the pulmonary hospitalizations.

Study results show while overall prevalence of non-tuberculosis mycobacteria lung disease is higher in women, prevalence increases for both sexes in the fifth or sixth decade of life, the scientists said.

The research appears in the journal Emerging Infectious Diseases.

Copyright 2009 by United Press International

Sourced and published by Henry Sapiecha 7th Oct 2009

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• We have in our faculty three of China’s top
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• The first to introduce Internal Gamma Knife or Nuclear
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• No excision required for the successful treatment of breast
  cancer
  
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Chinacancerhospital.com is dedicated to helping you to
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Sourced and published by Henry Sapiecha 7th Sept 2009

How can we avoid it?

Any involvement with  cystic fibrosis,? Then you understand and appreciate the difficulties that come with living with this disease.

So what exactly is it and how does it affect the body?

Cystic fibrosis is a chronic disease of the lungs and digestive system that is inherited and affects tens of thousands of people throughout the world ever year, both adults and children. This condition is caused by a defective gene which causes the body to produce thick, sticky mucus that clogs the lungs, making it difficult to breathe and causing life-threatening lung infections.

Cystic fibrosis symptoms

The symptoms of cystic fibrosis may include salty-tasting skin, persistent cough with phlegm, wheezing and shortness of breath, lung infections, poor growth and weight gain, difficulty with bowell movements, etc.

Statistics show that around 1,000 plus cases in the USA alone new cases of CF are discovered each year and most people are diagnosed before the age of two.

New testing techniques allow people to get a diagnosis at an earlier age, making it easier to seek proper treatment and maintenance of the condition.

Back about 50 years ago or before, people with cystic fibrosis would be expected to not make it into their teens. In fact, most children with the disease would not make it to elementary school. But today new advancements have made it possible for people with cystic fibrosis to live much longer lives and in many cases even live fairly normal lives, even with the condition.

Over 40% of the cystic fibrosis population today is over the age of 18 with the average life span reaching 37 years old. This may seem grim to some but when compared to the statistics of a few years ago, that is a major advancement. Advancements also include better treatments to help with CF in children and adults.

Cystic fibrosis treatment

Some treatments include causing cough to help loosen the mucus in the airways to increase breathing. There are also new therapies to help increase longevity and there are many organizations to help people with cystic fibrosis and their families. If you or someone in your family has cystic fibrosis, don’t be afraid to seek all the help you can get. You don’t have to live with this disease alone.

The more you learn about lung health and how to increase your lung health, the longer and happier you will live.

Learn this lesson quickly and add quality years to your life.

Published by Henry Sapiecha 24th August 2009

You Need to Know

IT IS EXTREMELY DANGEROUS

SARS is one of the most dangerous viruses on the face of the earth. SARS stands for Severe Acute Respiratory Syndrome

It infects the lungs. It is one of the most dangerous and contagious diseases on the face of the earth because it is most often fatal.

The first appearance of SARS was discovered in China recently  in November of late 2002.

Within an extremely important period of time, only six weeks, the SARS virus had spread throughout the entire world. It was mainly spread by international travelers that did not know they had contracted the disease.

During that period of time the World Health Organization or WHO, said that close to 8,000 people were infected. Along with those infections a total of eight hundred people died of the SARS syndrome. During this initial outbreak the entire world was in a complete panic. The world was concerned over the fact that the virus could have turned into a global pandemic. The good thing is that this pandemic never occurred and there has not been an outbreak of SARS since 2004.

SARS has many signs and symptoms that are associated with it.

When someone first contracts SARS they have a low fever.

This symptom usually does not start until ten days after the virus is contracted. Some of the most obvious signs of contraction of the SARS disease is a headache and muscle soreness. Another symptom that some people get is chills or discomfort.

Many people develop a small, dry cough after approximately five days. Some cases of SARS can develop into bad cases of pneumonia resulting in depleted oxygen levels in the blood. If you do have SARS you should stay home because you don’t want to start a global pandemic! After symptoms go away, you should still stay home for approximately ten days to ensure that your illness is gone.

Knowing when to see a doctor can sometimes be difficult. If you have any of the signs or symptoms of SARS you should immediately seek medical assistance. Part of saving yourself from SARS is catching it early. If you let SARS go untreated for a long period of time then you may find that it becomes fatal. There is little chance that SARS will ever turn into another global pandemic. It is still important to monitor people for symptoms of SARS because there is always a possibility that the illness will resurface once again.

Sourced and published by Henry Sapiecha 24th August 2009